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Asthma in the Workplace, Third Edition by David I. Bernstein, Moira Chan-Yeung, Jean-Luc Malo, I.

By David I. Bernstein, Moira Chan-Yeung, Jean-Luc Malo, I. Leonard Bernstein

Addressing each key component to occupational asthma-including ailment mechanisms, medical analysis, therapy, and different types of causative agents-this 3rd version offers a professional survey of the main influential advances and examine within the box. offering new and elevated chapters on genetics, environmental tracking, pathophysiology, pores and skin and pulmonary interactions, and the surveillance and prevention of occupational bronchial asthma, this advisor will stand on my own because the most recent resource at the subject.

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Extra info for Asthma in the Workplace, Third Edition

Sample text

TDI was found in the ventilation filters, the portal of entry into the storeroom. Provocation tests by painting with polyurethane resin plus TDI established TDI as the cause. These workers were subsequently compensated by the neighboring factory. The initial presenting patient was so sensitive that he had only to approach the area to develop severe asthma that day. In the case of amino-ethyl ethanolamine (37), a flux component for aluminum wire soldering was identified as the cause in a test consisting of three to four inhalations of the fumes, confirming the findings of Sterling (38).

S. states using surveillance guidelines for state health departments, California, Massachusetts, Michigan, and New Jersey, 1993–1995. Mor Mortal Wkly Rep CDC Surveill Summ 1999; 48:1–20. 18. Hnizdo E, Vallyathan V. Chronic obstructive pulmonary disease due to occupational exposure to silica dust: a review of epidemiological and pathological evidence. Occup Environ Med 2003; 60:237–243. 19. Petran M, Cocarla A, Baiescu M. Association between bronchial hyper-reactivity and exposure to silicon carbide.

There is as yet no explanation in general for isolated late reactions or the recurrent nocturnal reaction, in contrast to immediate and many dual reactions where specific IgE antibody is present as shown by skin and serological tests. The above list of agents and reactions to occupational-type tests will be expanded considerably in the chapters to follow, as will discussions and results of tests with other methods of exposure, because of their importance in establishing etiological diagnosis in OA.

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